Genetic Alcoholism

The study by South Korean scientists found that who can drink 90 times increase the probability of alcoholism genes.  South Korean research team who engaged in the studyteam has said that according to the genetic characteristics of different enzymes that breaks down alcohol in the consumption of alcohol can produce different effects, 87% of the alcoholic who is affected by this genetic influence, thus making Alcoholism the probability greatly enhanced.

This is the research team took three years to more than 1,000 patients with alcoholism

And the ordinary people of the clinical data and breaks down alcohol in hereditary enzyme analysis of the conclusions reached. The results published in recent issue of the International Journal of Genetics, “Human Molecular Genetics” on.

The research team of scientists said that generally speaking, ADH1B genes on the first 47 amino acids, histidine broken down into acetaldehyde by alcohol, speed, faster than the L-arginine were. ALDH2 genes in the first 48 amino acids, the glutamic acid decomposition of acetaldehyde into acetate were faster than ricin, quick, while the acetate decomposition poor people tend to cause various side effects of alcoholism.

The research team found that if the genes ADH1B the first 47 amino acids are arginine, ALDH2 genes in the first 48 amino acids are glutamic acid, the probability of alcoholism than in the two positions is histidine and castor-oil plant protein were higher than 90 times.

Also,United States National Institute on Alcohol Abuse and Alcoholism (NLAAA) recently reported to influence their susceptibility locus for alcohol research in the first major discovery. Genetics from the collaborative research group for Alcoholism (COGA) of the two studies have shown that alcoholism – susceptibility genes exist in many chromosomes.

One study examined 103 families prone to alcoholism in 607 member. As in the previous studies illustrate it, such people have a low P 3 amplitude (ampitude), this may indicate differences between the apparent difficulty to stimulate an awareness among the measured brain electrical activity. The researchers reported a lower P 3 amplitude and chromosome 2 and chromosome 6 close relationship between the genetic linkage and chromosome 5 and 13 with some evidence of links between. Scientists are convinced that they are beginning to address specific alcohol-dependent genes, but they think they need high-resolution map of the location to determine the amplitude with the low-P 3 which genes related to chromosomal region has led to the genetics contact.The second COGA study analyzed responses from 105 families, 987 people, these families is the occurrence of at least three immediate family members of alcohol dependent families. 291 people by examining the DNA markers, the researchers found that the chromosome 1 and 7, and alcohol dependence are closely related to link between the sensitivity of inadequate evidence. This effect is not reported in unaffected brothers of a chromosome No. 4 protective loci in the alcohol dehydrogenase gene cluster (Cluster) in the evidence. An examination of the independence of the southwestern United States population study shows that the chromosomal region similar to the protective role of the locus of this chromosome area give rise to alcohol produced a very good reaction to bad.Now, scientists will carry out the same crowd-depth study of such a link. In the next few years will be detachable out the exact genes. NLAAA Director Enoch Gordis, said the researchers carried out according to the results of these studies targeted preventive interventions, design and implementation of pharmacological treatment

United States National Institute on Alcohol Abuse and Alcoholism (NLAAA) recently reported to influence their susceptibility locus for alcohol research in the first major discovery. Genetics from the collaborative research group for Alcoholism (COGA) of the two studies have shown that alcoholism – susceptibility genes exist in many chromosomes. One study examined 103 families prone to alcoholism in 607 member. As in the previous studies illustrate it, such people have a low P 3 amplitude (ampitude), this may indicate differences between the apparent difficulty to stimulate an awareness among the measured brain electrical activity. The researchers reported a lower P 3 amplitude and chromosome 2 and chromosome 6 close relationship between the genetic linkage and chromosome 5 and 13 with some evidence of links between. Scientists are convinced that they are beginning to address specific alcohol-dependent genes, but they think they need high-resolution map of the location to determine the amplitude with the low-P 3 which genes related to chromosomal region has led to the genetics contact. The second COGA study analyzed responses from 105 families, 987 people, these families is the occurrence of at least three immediate family members of alcohol dependent families. 291 people by examining the DNA markers, the researchers found that the chromosome 1 and 7, and alcohol dependence are closely related to link between the sensitivity of inadequate evidence. This effect is not reported in unaffected brothers of a chromosome No. 4 protective loci in the alcohol dehydrogenase gene cluster (Cluster) in the evidence. An examination of the independence of the southwestern United States population study shows that the chromosomal region similar to the protective role of the locus of this chromosome area give rise to alcohol produced a very good reaction to bad. Now, scientists will carry out the same crowd-depth study of such a link. In the next few years will be detachable out the exact genes. NLAAA Director Enoch Gordis, said the researchers carried out according to the results of these studies targeted preventive interventions, design and implementation of pharmacological treatment

The study by South Korean scientists found that who can drink 90 times increase the probability of alcoholism genes.  South Korean research team who engaged in the studyteam has said that according to the genetic characteristics of different enzymes that breaks down alcohol in the consumption of alcohol can produce different effects, 87% of the alcoholic who is affected by this genetic influence, thus making Alcoholism the probability greatly enhanced.

This is the research team took three years to more than 1,000 patients with alcoholism

And the ordinary people of the clinical data and breaks down alcohol in hereditary enzyme analysis of the conclusions reached. The results published in recent issue of the International Journal of Genetics, “Human Molecular Genetics” on.

The research team of scientists said that generally speaking, ADH1B genes on the first 47 amino acids, histidine broken down into acetaldehyde by alcohol, speed, faster than the L-arginine were. ALDH2 genes in the first 48 amino acids, the glutamic acid decomposition of acetaldehyde into acetate were faster than ricin, quick, while the acetate decomposition poor people tend to cause various side effects of alcoholism.

The research team found that if the genes ADH1B the first 47 amino acids are arginine, ALDH2 genes in the first 48 amino acids are glutamic acid, the probability of alcoholism than in the two positions is histidine and castor-oil plant protein were higher than 90 times.

Also,United States National Institute on Alcohol Abuse and Alcoholism (NLAAA) recently reported to influence their susceptibility locus for alcohol research in the first major discovery. Genetics from the collaborative research group for Alcoholism (COGA) of the two studies have shown that alcoholism – susceptibility genes exist in many chromosomes.

One study examined 103 families prone to alcoholism in 607 member. As in the previous studies illustrate it, such people have a low P 3 amplitude (ampitude), this may indicate differences between the apparent difficulty to stimulate an awareness among the measured brain electrical activity. The researchers reported a lower P 3 amplitude and chromosome 2 and chromosome 6 close relationship between the genetic linkage and chromosome 5 and 13 with some evidence of links between. Scientists are convinced that they are beginning to address specific alcohol-dependent genes, but they think they need high-resolution map of the location to determine the amplitude with the low-P 3 which genes related to chromosomal region has led to the genetics contact.The second COGA study analyzed responses from 105 families, 987 people, these families is the occurrence of at least three immediate family members of alcohol dependent families. 291 people by examining the DNA markers, the researchers found that the chromosome 1 and 7, and alcohol dependence are closely related to link between the sensitivity of inadequate evidence. This effect is not reported in unaffected brothers of a chromosome No. 4 protective loci in the alcohol dehydrogenase gene cluster (Cluster) in the evidence. An examination of the independence of the southwestern United States population study shows that the chromosomal region similar to the protective role of the locus of this chromosome area give rise to alcohol produced a very good reaction to bad.Now, scientists will carry out the same crowd-depth study of such a link. In the next few years will be detachable out the exact genes. NLAAA Director Enoch Gordis, said the researchers carried out according to the results of these studies targeted preventive interventions, design and implementation of pharmacological treatment

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